![]() ![]() Non-iatrogenic traumatic causes involve sharp injuries, intraocular foreign bodies, or blunt injuries causing increased intraocular pressure and inside out injury leading to open globe injury (rupture). ![]() Occasionally, complete sloughing of strangulated tissue within the suture loop may occur. Once communication between the posterior wound gap and the anterior wound defect occurs (subsequent to tissue necrosis from tight sutures), anterior aqueous fluid may egress vitreous incarceration may also occur, producing the vitreous wick. ![]() Tightly compressed corneal wound edges may demonstrate puckering and also may lead to enlargement of suture tracts, promoting tissue necrosis within the suture loop. Poor suturing technique is implicated as a major factor for wound breakdown. Ĭorneal wound healing has been documented to be slower on the endothelial side (inner layers). Microscopic wound breakdown has been hypothesized as the “point of no return” for the development of vitreous wick syndrome-a point emphasized by Ruiz and Teeters in their initial description. Vitreous wick syndrome of iatrogenic origin usually follows anterior-segment surgery, though it may also follow sub Tenon injection and muscle surgery if sclera is perforated. Vitreous wick syndrome develops in the setting of trauma, either iatrogenic or non-iatrogenic. No gender predisposition has been identified, and the condition has no apparent racial predilection. No age predisposition has been documented for this syndrome.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |